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PI3K/AKT signaling in the APP/PS1 mouse model

As such, zinc, which is able to function both as a signaling mediator and neurotoxin, has been implicated in cerebral ischemia

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Although this study suggested that extracellular zinc levels drop during ischemia, more recent micro-dialysis studies suggest that while a temporal derangement in extracellular zinc levels during ischemia occurs, levels are increasing rather than decreasing