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Plasma and tissue GSH levels decline with age, while markers of oxidative damage increase, creating a "redox imbalance" implicated in the pathogenesis of age-related diseases [2][3][10][11]

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Compared with controls, patients who received GLP-1s had a lower risk for all-cause mortality (HR = 0.29

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However, in vitro, zinc inhibited superoxide production by macrophages derived from septic rats [240]

Combination treatment of SGLT2 inhibitors and GLP-1 receptor agonists: symbiotic effects on metabolism and cardiorenal risk