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[21], who reported significant improvements in comorbidities following SG in youth

Chronic ER stress, resulting from impaired ERAD, triggers the UPR, which, if unresolved, can lead to cardiomyocyte apoptosis and heart failure

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Cu 2+ induces a conformational change into a more compact structure containing -helices that resists HDX and is protected from enzymatic hydrolysis by insulin degrading enzyme (IDE)