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2.2 Neurotoxic mechanisms METH induces neurotoxicity via a combination of oxidative stress, mitochondrial dysfunction, excitotoxicity, and neuroinflammation

Immunofluorescence imaging revealed that FITC-labeled exendin 939 was bound to TRPV1 on the cell surface in CHO K1 cells expressing human TRPV1 but not in nave CHO K1 cells (Fig

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This is done through a process called glycogenolysis, which occurs when glucagon in the liver stimulates the conversion of stored glycogen to glucose

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